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AKT kinases as therapeutic targets

Owing to their intrinsic disorder and poor sequence conservation, TADs remain difficult to predict in silico from primary sequences, even with recent large-scale ED catalogs and deep-learning tools developed in yeast, fly, human, and plant systems 42,68,69,77,78,79,80

Further exploration revealed that the downregulation of HMGB1 expression effectively inhibited the NF-B/p65 and its binding activity to DNA, thereby reducing the expression of vascular endothelial growth factor C (VEGF-C) and impeding tumor angiogenesis

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