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Studies using mouse models have confirmed that elevated HO-1 not only exacerbates the inflammatory response in macrophage-derived foam cells but also induces ferroptosis, resulting in a significantly increased plaque burden (77)
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The biggest difference in the semaglutide vs tirzepatide discussion is that Semaglutide works only on GLP-1 receptors, while Tirzepatide activates both GLP-1 and GIP receptors
