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These effects may be due to vagus nerve stimulation of adrenal catecholamine production (Torres-Rosas et al., 2014), as patients with sepsis often show adrenal insufficiency (Marik and Zaloga, 2003)

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Mitochondrial dysfunctions in ASD individuals could also result from various risk factors, both endogenous and exogenous, including toxins, immune stimulation, drugs, and metabolic abnormalities [17]

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