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[] However, the source of TNFSF14 in this study is unclear
Immunotherapy approaches targeting immune checkpoint inhibitors such as PD-1/PD-L1 and CTLA-4 have been ongoing in GBM treatment
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NADPH, a key intracellular reductant, is required for glutathione system and other ROS scavengers to maintain the redox homeostasis (115)
