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217 The use of a specific cell line to quantify the activation of the redox-sensitive transcription factor, nuclear factor (erythroid-derived 2)-like 2 (Nrf2), allowed to demonstrate that methotrexate also reduces the activation of the Nrf2-dependent intracellular redox signalling pathway

The intriguing possibility of targeting the IGF axis for treating cardiovascular and metabolic disorders remains uncertain due to adverse effects observed in pre-clinical and clinical studies

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We therefore compared these methodologies in an animal model of pharmacologic glutathione depletion, to determine if ascorbate-derived HP MR or PET probes could detect the modulation of redox capacity