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Description
PGC-1 expression and activity are controlled by p38 MAPK, and a CAMK inhibitor prevented p38 activation in response to increased cytosolic Ca 2+ , demonstrating that p38 MAPK is a downstream target of CAMK [77,78,79,80]
under the accession number of PRJNA1002561
This supports the concept that PPAR does not affect APAP metabolism [26]
31 pH-responsive materials take advantage of the unique pH characteristics of the gastric environment
