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Although the causes underlying SIR remain to be identified, tissue contents of glutathione or precursors may be among the factors linking sulfur nutrition to the responses of plants to fungal and viral infection (Gullner et al., 1999

Mechanistically, EDCs can act as agonists or antagonists at estrogen receptors (ER and ER), androgen receptors (ARs), or thyroid hormone receptors (TRs), thereby altering receptor conformation, recruitment of coactivators or corepressors, and downstream transcriptional activity [72,73]

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Interestingly, the deletion of gliK also led to the elevation of ergothioneine [110], which is also an intracellular antioxidant
